Resurrection! The Cause of Cancer Re-Discovered


Resurrection! The Cause of Cancer Re-Discovered

or What The Literature Says About the Cause, Prevention and Treatment of Cancer

a review by the author, E. Donald Wilson, B.Sc.

There are literally hundreds of books about ways to treat or beat cancer. Can the market stand another one?

Only if it has something different to offer. This book does just that!

The author had cancer 18 years ago and went through the horrible experience of radiation and chemo. But he also started reading books and technical papers, etc., and he made some startling discoveries, such as:

The cause of cancer was discovered early in the 20th century and has been re-discovered independently by upwards of 10 investigators.
The cause is a microflora with which we are all born. They change form and cause disease. Over 50 doctors or scientists have reported finding a microbe associated with cancer.
The conventional medical community (CMC) has chosen to ignore the microflora. Why? Because over 100 years ago Louis Pasteur said the microflora did not exist. Pasteur’s claims are still being taught in medical schools.
So the CMC does not know what causes cancer except for leukemia and maybe one or two other relatively minor types of cancer that are caused by a virus.
The CMC has been working on a theory for many years that cancer is caused by gene mutation which, in turn, results in cell mutation and malignancy. The exact process has yet to be demonstrated.
Several alternative therapies for the treatment of cancer have been developed over the years, but none has been subjected to a scientifically designed clinical trial.
Despite the expenditure of over 30 billion dollars for research on the cause and treatment of cancer, the overall mortality from cancer increased steadily for 30 or more years in Canada and the United States, though it has decreased slightly in the last few years.
In bringing these findings to the attention of the reader, the book presents the best-ever summary of the history of the development of the microbial theory of cancer. It presents the informed opinions of a researcher that provide a logical explanation of why the CMC has produced little of benefit to the cancer victim. It cites the stories of six medical doctors who cured their own cancer using alternative therapies. And it includes a priceless article, the result of an extensive search of the literature on the question of whether organic foods are really superior. The results and conclusions are startling!

This book provides the cancer victim, and any person worried about being diagnosed with cancer, with an easy-to-read bonanza of information on its prevention and many options for its treatment. This will be a godsend, especially for the newly diagnosed patient. There are so many books, some of them several hundreds of pages long, while the patient wants more or less instant information. Here is the story in less than 100 pages, not including the appendices, glossary, references and index. (To order this book see Order Form enclosed.)

This is Bonnie again: In this book I found an answer to a question I have had since 1982!

Question: "Why did the Gerson Clinic serve patients, twice a day, a ½ glass of raw, organic calves liver juice — the staff ground and pressed the liver fresh twice a day — mixed with a ½ glass of carrot juice?" (They now use desiccated liver capsules instead.) (Please see our order form for desiccated liver tablets.)

Answer: Abscisic Acid

"Abscisic acid is a supplement you will not find in a health food store. Based on her research, Dr. Livingston-Wheeler determined that abscisic acid neutralizes the effect of chorionic gonadotropin, the hormone that protects the fetus from being rejected, and she classed it as her prime supplement in treating cancer. Livingston-Wheeler discovered that cancer cells (or the microbes associated with them) produce a substance very similar to chorionic gonadotropin. Abscisic acid is a close relative of Vitamin A. In her book, The Conquest of Cancer; Livingston-Wheeler describes how you can make abscisic acid.

"Add one teaspoon of raw liver powder to an 8 oz glass of carrot juice (must be organic). Stir and let stand for fifteen to twenty minutes and then drink it.

"The enzymes in the liver powder convert some of the Vitamin A in the carrot juice into abscisic acid."

Abscisic Acid (ABA)

During the fifties were the shedding of fruits and leaves, also called abscission, and the dormancy of buds intensely studied. This lead to the discovery of a hormone called abscisic acid. It showed that this substance is, too, wide-spread in the plant kingdom. Cotton fruits became a suitable source for the isolation amounts large enough to elucidate the chemical structure (F. F. ADDINCOTT and collaborators 1961, 1963, 1969, B. W. MILBORROW, 1967).

Abscisic acid (ABA) is identical with a substance that causes bud dormancy in wooden perennial plants. It was therefore at first also called dormin. In maple and birch buds causes the change from long-day to short-day conditions a marked increase in the activity of dormin (=ABA) and consequently stops the growth of buds.

ABA-containing maple and birch leaf extracts from plants grown under short-day conditions inhibit leaf growth and induce even in fast growing shoots dormant buds. When the formula of ABA was known the production of a number of derivatives began none of which attained the effect of ABA.

In some plant tissues (especially in young shoots) occurs a related compound called xanthoxine.

Whether xanthoxine is an intermediate of the ABA-biosynthesis or whether it is an independent product remains unknown. The structure indicates that both ABA and xanthoxine are terpene derivatives. This was proven when it could be shown that radioactively labelled mevalonic acid is integrated into ABA though it does not elucidate which intermediates are produced. Two alternative biosyntheses have been discussed:

ABA is a degradation product of xanthophyll (especially of violaxanthin).
ABA is produced from a C15 precursor using a separate pathway and is thus independent from the carotenoid/xanthophyll metabolism.
The first idea seemed initially more plausible since the structures of xanthophylls and ABA correspond to a large degree. In vitro occurs conversion only upon exposure to strong light and with an extremely low yield, though. This and supplementing in vitro observations called the first assumption into question again.

Biological activities: Inhibition of growth and maintenance of the dormancy of buds are the most striking effects of ABA. ABA activity alone is not enough to keep the dormancy of buds up in the long term though. After sprouting falls the concentration of ABA in the buds significantly whilst it remains unknown whether the decrease is caused by a lack of supply, enforced breakdown or modifications (like glycosylation). On the contrary rises the ABA level during seed and fruit production.

ABA is an efficient inhibitor of germination and occurs in high concentrations in dormant seeds. Just as in sprouting buds decreases its content also during seed germination, an indication that germination is controlled by an equilibrium of auxin(s), gibberellin(s), and cytokinin(s) on one and ABA on the other hand. The role ABA has during the abscission of fruits and leaves is largely unknown. Though both cases seem to be governed by similar mechanisms has ABA nearly no effect on the abscission of leaves while it shows a clear effect on fruit abscission. Moreover was a regulating effect of abscisic acid on the water balance observed.

As soon as the water supply of cut wheat leaf blades is interrupted and the cell turgor decreases raises the concentration of ABA forty-fold within four hours. Comparable data have been collected for other plant species, too. These effects were also observed in rooted shoots. A water loss of 5 – 10 percent (of the green weight) was sufficient to increase the ABA level. The raise is based on a new synthesis and not on the release of an inactive state as could be shown by W. MILBORROW. The concentration of ABA remains high if the plant’s situation improves slightly or even decreases dramatically. It induces the stomata to close thus inhibiting further loss of water.

Though it cannot be said too much about the mode of action does it seem certain that ABA inhibits the guard cells’ uptake of potassium ions. Potassium ions are essential for the opening mechanism of the guard cells (K. RASCHKE, 1975).

ABA reverses the effect of growth-stimulating hormones (auxin, gibberellins, cytokinin) in several tissues. The synthesis of hydroxylases within germinating wheat seeds, for example, does not take place after the application of ABA.

In summary can the importance of the ABA effect be interpreted as an effector that has the ability to close down certain parts of the plant metabolism for a period of time. Since ABA is easily removed from tissues, is its effect reversible. An example is the inhibition of seed germination in berries (like tomatoes). The germination does not occur even though the seeds are in a humid surrounding. When isolated and transferred to a normal, humid milieu do they immediately start to germinate. The failure of the inhibition of seed germination leads usually to vivipary.

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